Berger disease: therapeutic algorithm proposal and chronic renal failure predictive factors.
Autor(a) principal: | |
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Data de Publicação: | 2003 |
Outros Autores: | , |
Tipo de documento: | Artigo |
Idioma: | por |
Título da fonte: | Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos) |
Texto Completo: | https://www.actamedicaportuguesa.com/revista/index.php/amp/article/view/1185 |
Resumo: | Berger disease is described as a glomerulonephritis by anti-IgA and anti-IgG immune complexes and it has become the most common form of glomerulonephritis, affecting up to 1,3% of the population, most often affecting young adult males. Berger disease etiopathogenesis is still unknown, being well recognized its temporal associations with bacterial and viral infections, as well with genetic factors which lead some authors to suggest the existence of a founder effect. Characteristically IgA nephropathy presents intermittent macroscopic hematuria following mucosal infections, nonnephrotic proteinuria and persistent microscopic hematuria. A renal biopsy should be performed in patients in whom this diagnosis is suspected, particularly if there is persistent proteinuria. IgA nephropathy was initially considered a benign disease, but long term follow up studies have shown the course to be slowly progressive with up to 50% of the patients developing terminal renal failure. The authors propose themselves a therapeutic algoritm, as well as clinical indicators of evolutive course. |
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Berger disease: therapeutic algorithm proposal and chronic renal failure predictive factors.Doença de Berger: proposta de algoritmo terapêutico e factorespreditivos de insuficiência renal crónica.Berger disease is described as a glomerulonephritis by anti-IgA and anti-IgG immune complexes and it has become the most common form of glomerulonephritis, affecting up to 1,3% of the population, most often affecting young adult males. Berger disease etiopathogenesis is still unknown, being well recognized its temporal associations with bacterial and viral infections, as well with genetic factors which lead some authors to suggest the existence of a founder effect. Characteristically IgA nephropathy presents intermittent macroscopic hematuria following mucosal infections, nonnephrotic proteinuria and persistent microscopic hematuria. A renal biopsy should be performed in patients in whom this diagnosis is suspected, particularly if there is persistent proteinuria. IgA nephropathy was initially considered a benign disease, but long term follow up studies have shown the course to be slowly progressive with up to 50% of the patients developing terminal renal failure. The authors propose themselves a therapeutic algoritm, as well as clinical indicators of evolutive course.Berger disease is described as a glomerulonephritis by anti-IgA and anti-IgG immune complexes and it has become the most common form of glomerulonephritis, affecting up to 1,3% of the population, most often affecting young adult males. Berger disease etiopathogenesis is still unknown, being well recognized its temporal associations with bacterial and viral infections, as well with genetic factors which lead some authors to suggest the existence of a founder effect. Characteristically IgA nephropathy presents intermittent macroscopic hematuria following mucosal infections, nonnephrotic proteinuria and persistent microscopic hematuria. A renal biopsy should be performed in patients in whom this diagnosis is suspected, particularly if there is persistent proteinuria. IgA nephropathy was initially considered a benign disease, but long term follow up studies have shown the course to be slowly progressive with up to 50% of the patients developing terminal renal failure. The authors propose themselves a therapeutic algoritm, as well as clinical indicators of evolutive course.Ordem dos Médicos2003-08-31info:eu-repo/semantics/publishedVersioninfo:eu-repo/semantics/articleapplication/pdfhttps://www.actamedicaportuguesa.com/revista/index.php/amp/article/view/1185oai:ojs.www.actamedicaportuguesa.com:article/1185Acta Médica Portuguesa; Vol. 16 No. 4 (2003): July-August; 261-6Acta Médica Portuguesa; Vol. 16 N.º 4 (2003): Julho-Agosto; 261-61646-07580870-399Xreponame:Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos)instname:Agência para a Sociedade do Conhecimento (UMIC) - FCT - Sociedade da Informaçãoinstacron:RCAAPporhttps://www.actamedicaportuguesa.com/revista/index.php/amp/article/view/1185https://www.actamedicaportuguesa.com/revista/index.php/amp/article/view/1185/837Pessegueiro, PedroBarata, ConceiçãoCorreia, Joséinfo:eu-repo/semantics/openAccess2022-12-20T10:57:35Zoai:ojs.www.actamedicaportuguesa.com:article/1185Portal AgregadorONGhttps://www.rcaap.pt/oai/openaireopendoar:71602024-03-19T16:17:00.983508Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos) - Agência para a Sociedade do Conhecimento (UMIC) - FCT - Sociedade da Informaçãofalse |
dc.title.none.fl_str_mv |
Berger disease: therapeutic algorithm proposal and chronic renal failure predictive factors. Doença de Berger: proposta de algoritmo terapêutico e factorespreditivos de insuficiência renal crónica. |
title |
Berger disease: therapeutic algorithm proposal and chronic renal failure predictive factors. |
spellingShingle |
Berger disease: therapeutic algorithm proposal and chronic renal failure predictive factors. Pessegueiro, Pedro |
title_short |
Berger disease: therapeutic algorithm proposal and chronic renal failure predictive factors. |
title_full |
Berger disease: therapeutic algorithm proposal and chronic renal failure predictive factors. |
title_fullStr |
Berger disease: therapeutic algorithm proposal and chronic renal failure predictive factors. |
title_full_unstemmed |
Berger disease: therapeutic algorithm proposal and chronic renal failure predictive factors. |
title_sort |
Berger disease: therapeutic algorithm proposal and chronic renal failure predictive factors. |
author |
Pessegueiro, Pedro |
author_facet |
Pessegueiro, Pedro Barata, Conceição Correia, José |
author_role |
author |
author2 |
Barata, Conceição Correia, José |
author2_role |
author author |
dc.contributor.author.fl_str_mv |
Pessegueiro, Pedro Barata, Conceição Correia, José |
description |
Berger disease is described as a glomerulonephritis by anti-IgA and anti-IgG immune complexes and it has become the most common form of glomerulonephritis, affecting up to 1,3% of the population, most often affecting young adult males. Berger disease etiopathogenesis is still unknown, being well recognized its temporal associations with bacterial and viral infections, as well with genetic factors which lead some authors to suggest the existence of a founder effect. Characteristically IgA nephropathy presents intermittent macroscopic hematuria following mucosal infections, nonnephrotic proteinuria and persistent microscopic hematuria. A renal biopsy should be performed in patients in whom this diagnosis is suspected, particularly if there is persistent proteinuria. IgA nephropathy was initially considered a benign disease, but long term follow up studies have shown the course to be slowly progressive with up to 50% of the patients developing terminal renal failure. The authors propose themselves a therapeutic algoritm, as well as clinical indicators of evolutive course. |
publishDate |
2003 |
dc.date.none.fl_str_mv |
2003-08-31 |
dc.type.status.fl_str_mv |
info:eu-repo/semantics/publishedVersion |
dc.type.driver.fl_str_mv |
info:eu-repo/semantics/article |
format |
article |
status_str |
publishedVersion |
dc.identifier.uri.fl_str_mv |
https://www.actamedicaportuguesa.com/revista/index.php/amp/article/view/1185 oai:ojs.www.actamedicaportuguesa.com:article/1185 |
url |
https://www.actamedicaportuguesa.com/revista/index.php/amp/article/view/1185 |
identifier_str_mv |
oai:ojs.www.actamedicaportuguesa.com:article/1185 |
dc.language.iso.fl_str_mv |
por |
language |
por |
dc.relation.none.fl_str_mv |
https://www.actamedicaportuguesa.com/revista/index.php/amp/article/view/1185 https://www.actamedicaportuguesa.com/revista/index.php/amp/article/view/1185/837 |
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info:eu-repo/semantics/openAccess |
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openAccess |
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application/pdf |
dc.publisher.none.fl_str_mv |
Ordem dos Médicos |
publisher.none.fl_str_mv |
Ordem dos Médicos |
dc.source.none.fl_str_mv |
Acta Médica Portuguesa; Vol. 16 No. 4 (2003): July-August; 261-6 Acta Médica Portuguesa; Vol. 16 N.º 4 (2003): Julho-Agosto; 261-6 1646-0758 0870-399X reponame:Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos) instname:Agência para a Sociedade do Conhecimento (UMIC) - FCT - Sociedade da Informação instacron:RCAAP |
instname_str |
Agência para a Sociedade do Conhecimento (UMIC) - FCT - Sociedade da Informação |
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RCAAP |
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RCAAP |
reponame_str |
Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos) |
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Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos) |
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Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos) - Agência para a Sociedade do Conhecimento (UMIC) - FCT - Sociedade da Informação |
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