Acute Effects Of Smoking On Blood Pressure And Heart Rate In Young Adults

Detalhes bibliográficos
Autor(a) principal: Dias Costa, Joana
Data de Publicação: 2023
Outros Autores: Valentim, Bruno, Coelho, Patrícia, Rodrigues, Francisco, Pires, Joana
Tipo de documento: Artigo
Idioma: por
Título da fonte: Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos)
Texto Completo: https://doi.org/10.58043/rphrc.74
Resumo: Introduction: Tobacco (Nicotiana tabacum) is a psychoactive plant, whose main chemical is nicotine, which is extremely addictive. Smokers have twice the risk of developing cardiovascular disease compared to non-smokers. At the level of the cardiovascular system, nicotine being a sympatheticmimetic substance, it promotes the release of catecholamines, epinephrine and norepinephrine, activated by the sympathetic nervous system. This activation of the sympathetic nervous system is responsible for increased heart rate (HR) and systolic blood pressure (SBP) and diastolic blood pressure (DBP). The study aims to understand whether tobacco consumption has an acute impact on BP and HR values. Material and methods: The present study was observational, cross-sectional and analytical. The sample consisted of 28 young adults, of both sexes, between 18 and 30 years of age. BP and HR were evaluated using the diagnostic examination of ambulatory blood pressure monitoring, Welch Allyn ABPM6100. BP and HR were evaluated before, during and after smoking. Results: There was a significant increase in SBP, DBP and HR values during smoking in both cigarettes, with maximum mean values (1st cigarette: SBP 133.21 mmHg, DBP 81.57 mmHg and HR 98.93 bpm; 2nd cigarette: SBP 132.82 mmHg, DBP 78.89 mmHg and HR 92.46 bpm). After smoking, there was a significant decrease in BP and HR at 15 minutes of recovery, with minimum mean values in the 3rd evaluation (1st cigarette: SBP 126.43 mmHg, DBP 75.79 mmHg and HR 83.82 bpm; 2nd cigarette: SBP 126.43 mmHg, DBP 76.14 mmHg and HR 83.79 bpm). Regarding the values referring to the moment before smoking each cigarette, it was observed that SBP, DBP were higher in the 2nd cigarette compared to the 1st cigarette. Discussion: During the act of smoking, in both cigarettes, there were significant differences for SBP, DBP and HR, when compared the moment before smoking with the 1st evaluation at the time of smoking. The nicotine present in cigarettes promotes the release of neurotransmitters, norepinephrine, and epinephrine, activated by the sympathetic nervous system, being responsible for the increase in BP and HR. Conclusion: The beginning of smoking is associated with an increase in BP and HR values, decreasing after smoking.
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spelling Acute Effects Of Smoking On Blood Pressure And Heart Rate In Young AdultsOs Efeitos Agudos Do Consumo Do Tabaco Na Pressão Arterial E Frequência Cardíaca Em Jovens AdultosTabagismoPressão ArterialFrequência CardíacaHipertensão ArterialSmokingBlood PressureHeart RateHypertensionIntroduction: Tobacco (Nicotiana tabacum) is a psychoactive plant, whose main chemical is nicotine, which is extremely addictive. Smokers have twice the risk of developing cardiovascular disease compared to non-smokers. At the level of the cardiovascular system, nicotine being a sympatheticmimetic substance, it promotes the release of catecholamines, epinephrine and norepinephrine, activated by the sympathetic nervous system. This activation of the sympathetic nervous system is responsible for increased heart rate (HR) and systolic blood pressure (SBP) and diastolic blood pressure (DBP). The study aims to understand whether tobacco consumption has an acute impact on BP and HR values. Material and methods: The present study was observational, cross-sectional and analytical. The sample consisted of 28 young adults, of both sexes, between 18 and 30 years of age. BP and HR were evaluated using the diagnostic examination of ambulatory blood pressure monitoring, Welch Allyn ABPM6100. BP and HR were evaluated before, during and after smoking. Results: There was a significant increase in SBP, DBP and HR values during smoking in both cigarettes, with maximum mean values (1st cigarette: SBP 133.21 mmHg, DBP 81.57 mmHg and HR 98.93 bpm; 2nd cigarette: SBP 132.82 mmHg, DBP 78.89 mmHg and HR 92.46 bpm). After smoking, there was a significant decrease in BP and HR at 15 minutes of recovery, with minimum mean values in the 3rd evaluation (1st cigarette: SBP 126.43 mmHg, DBP 75.79 mmHg and HR 83.82 bpm; 2nd cigarette: SBP 126.43 mmHg, DBP 76.14 mmHg and HR 83.79 bpm). Regarding the values referring to the moment before smoking each cigarette, it was observed that SBP, DBP were higher in the 2nd cigarette compared to the 1st cigarette. Discussion: During the act of smoking, in both cigarettes, there were significant differences for SBP, DBP and HR, when compared the moment before smoking with the 1st evaluation at the time of smoking. The nicotine present in cigarettes promotes the release of neurotransmitters, norepinephrine, and epinephrine, activated by the sympathetic nervous system, being responsible for the increase in BP and HR. Conclusion: The beginning of smoking is associated with an increase in BP and HR values, decreasing after smoking.Introdução: O tabaco (Nicotiana tabacum) é uma planta psicoativa, cuja principal substância química é a nicotina, a qual é extremamente viciante. Os fumadores apresentam o dobro do risco de desenvolvimento de doenças cardiovasculares comparativamente com os não fumadores. Ao nível do sistema cardiovascular, sendo a nicotina uma substância simpaticomimética, promove a libertação de catecolaminas, epinefrina e norepinefrina, ativadas pelo sistema nervoso simpático. Esta ativação do sistema nervoso simpático é responsável pelo aumento da frequência cardíaca (FC) e pressão arterial sistólica (PAS) e pressão arterial diastólica (PAD). O estudo tem como objetivo perceber se o consumo do tabaco tem um impacto agudo nos valores da PA e FC. Material e métodos: O presente estudo foi do tipo observacional, transversal e analítico. A amostra é constituída por 28 jovens adultos, de ambos os sexos, entre os 18 e 30 anos. A PA e FC foram avaliadas com recurso ao exame de diagnóstico de monitorização ambulatória da pressão arterial, Welch Allyn ABPM6100. A PA e FC foram avaliadas antes, durante e após o ato de fumar. Resultados: Registou-se um aumento significativo dos valores da PAS, PAD e FC, durante o ato de fumar, nos dois cigarros, com valores médios máximos (1o cigarro: PAS 133,21 mmHg, PAD 81,57 mmHg e FC 98,93 bpm; 2o cigarro: PAS 132,82 mmHg, PAD 78,89 mmHg e FC 92,46 bpm). Após o ato de fumar, verificou-se uma diminuição significativa da PA e FC aos 15 minutos de recuperação, com valores médios mínimos na 3a avaliação (1o cigarro: PAS 126,43 mmHg, PAD 75,79 mmHg e FC 83,82 bpm; 2o cigarro: PAS 126,43 mmHg, PAD 76,14 mmHg e FC 83,79 bpm). Relativamente aos valores referentes ao momento antes de fumar cada cigarro, observou-se que a PAS, PAD foram superiores no 2o cigarro relativamente ao 1o cigarro. Discussão: Durante o ato de fumar, nos dois cigarros, verificaram-se diferenças significativas para a PAS, PAD e FC, quando comparados o momento antes de fumar com a 1a avaliação no ato de fumar. A nicotina presente no cigarro promove a libertação de neurotransmissores, norepinefrina e epinefrina, ativados pelo sistema nervoso simpático, sendo responsável pelo aumento da PA e FC. Conclusão: O início do ato de fumar está associado a um aumento dos valores da PA e FC, diminuindo após fumar um cigarro.Revista Portuguesa de Hipertensão e Risco Cardiovascular2023-04-16info:eu-repo/semantics/publishedVersioninfo:eu-repo/semantics/articleapplication/pdfhttps://doi.org/10.58043/rphrc.74https://doi.org/10.58043/rphrc.74Revista Portuguesa de Hipertensão e Risco Cardiovascular; N.º 94 (2023): Março / Abril; 14-221646-8287reponame:Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos)instname:Agência para a Sociedade do Conhecimento (UMIC) - FCT - Sociedade da Informaçãoinstacron:RCAAPporhttps://revistahipertensao.pt/index.php/rh/article/view/74https://revistahipertensao.pt/index.php/rh/article/view/74/72Direitos de Autor (c) 2023 Joana Dias Costa, Bruno Valentim, Patrícia Coelho, Francisco Rodrigues, Joana Piresinfo:eu-repo/semantics/openAccessDias Costa, JoanaValentim, BrunoCoelho, PatríciaRodrigues, FranciscoPires, Joana2024-02-03T07:36:51Zoai:ojs.revistahipertensao.pt:article/74Portal AgregadorONGhttps://www.rcaap.pt/oai/openaireopendoar:71602024-03-19T17:50:06.178128Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos) - Agência para a Sociedade do Conhecimento (UMIC) - FCT - Sociedade da Informaçãofalse
dc.title.none.fl_str_mv Acute Effects Of Smoking On Blood Pressure And Heart Rate In Young Adults
Os Efeitos Agudos Do Consumo Do Tabaco Na Pressão Arterial E Frequência Cardíaca Em Jovens Adultos
title Acute Effects Of Smoking On Blood Pressure And Heart Rate In Young Adults
spellingShingle Acute Effects Of Smoking On Blood Pressure And Heart Rate In Young Adults
Dias Costa, Joana
Tabagismo
Pressão Arterial
Frequência Cardíaca
Hipertensão Arterial
Smoking
Blood Pressure
Heart Rate
Hypertension
title_short Acute Effects Of Smoking On Blood Pressure And Heart Rate In Young Adults
title_full Acute Effects Of Smoking On Blood Pressure And Heart Rate In Young Adults
title_fullStr Acute Effects Of Smoking On Blood Pressure And Heart Rate In Young Adults
title_full_unstemmed Acute Effects Of Smoking On Blood Pressure And Heart Rate In Young Adults
title_sort Acute Effects Of Smoking On Blood Pressure And Heart Rate In Young Adults
author Dias Costa, Joana
author_facet Dias Costa, Joana
Valentim, Bruno
Coelho, Patrícia
Rodrigues, Francisco
Pires, Joana
author_role author
author2 Valentim, Bruno
Coelho, Patrícia
Rodrigues, Francisco
Pires, Joana
author2_role author
author
author
author
dc.contributor.author.fl_str_mv Dias Costa, Joana
Valentim, Bruno
Coelho, Patrícia
Rodrigues, Francisco
Pires, Joana
dc.subject.por.fl_str_mv Tabagismo
Pressão Arterial
Frequência Cardíaca
Hipertensão Arterial
Smoking
Blood Pressure
Heart Rate
Hypertension
topic Tabagismo
Pressão Arterial
Frequência Cardíaca
Hipertensão Arterial
Smoking
Blood Pressure
Heart Rate
Hypertension
description Introduction: Tobacco (Nicotiana tabacum) is a psychoactive plant, whose main chemical is nicotine, which is extremely addictive. Smokers have twice the risk of developing cardiovascular disease compared to non-smokers. At the level of the cardiovascular system, nicotine being a sympatheticmimetic substance, it promotes the release of catecholamines, epinephrine and norepinephrine, activated by the sympathetic nervous system. This activation of the sympathetic nervous system is responsible for increased heart rate (HR) and systolic blood pressure (SBP) and diastolic blood pressure (DBP). The study aims to understand whether tobacco consumption has an acute impact on BP and HR values. Material and methods: The present study was observational, cross-sectional and analytical. The sample consisted of 28 young adults, of both sexes, between 18 and 30 years of age. BP and HR were evaluated using the diagnostic examination of ambulatory blood pressure monitoring, Welch Allyn ABPM6100. BP and HR were evaluated before, during and after smoking. Results: There was a significant increase in SBP, DBP and HR values during smoking in both cigarettes, with maximum mean values (1st cigarette: SBP 133.21 mmHg, DBP 81.57 mmHg and HR 98.93 bpm; 2nd cigarette: SBP 132.82 mmHg, DBP 78.89 mmHg and HR 92.46 bpm). After smoking, there was a significant decrease in BP and HR at 15 minutes of recovery, with minimum mean values in the 3rd evaluation (1st cigarette: SBP 126.43 mmHg, DBP 75.79 mmHg and HR 83.82 bpm; 2nd cigarette: SBP 126.43 mmHg, DBP 76.14 mmHg and HR 83.79 bpm). Regarding the values referring to the moment before smoking each cigarette, it was observed that SBP, DBP were higher in the 2nd cigarette compared to the 1st cigarette. Discussion: During the act of smoking, in both cigarettes, there were significant differences for SBP, DBP and HR, when compared the moment before smoking with the 1st evaluation at the time of smoking. The nicotine present in cigarettes promotes the release of neurotransmitters, norepinephrine, and epinephrine, activated by the sympathetic nervous system, being responsible for the increase in BP and HR. Conclusion: The beginning of smoking is associated with an increase in BP and HR values, decreasing after smoking.
publishDate 2023
dc.date.none.fl_str_mv 2023-04-16
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dc.identifier.uri.fl_str_mv https://doi.org/10.58043/rphrc.74
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dc.publisher.none.fl_str_mv Revista Portuguesa de Hipertensão e Risco Cardiovascular
publisher.none.fl_str_mv Revista Portuguesa de Hipertensão e Risco Cardiovascular
dc.source.none.fl_str_mv Revista Portuguesa de Hipertensão e Risco Cardiovascular; N.º 94 (2023): Março / Abril; 14-22
1646-8287
reponame:Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos)
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