Prenatally programmed hypertension: role of maternal diabetes

Detalhes bibliográficos
Autor(a) principal: Gomes,G.N.
Data de Publicação: 2011
Outros Autores: Gil,F.Z.
Tipo de documento: Artigo
Idioma: eng
Título da fonte: Brazilian Journal of Medical and Biological Research
Texto Completo: http://old.scielo.br/scielo.php?script=sci_arttext&pid=S0100-879X2011000900011
Resumo: Epidemiological and experimental studies have led to the hypothesis of the fetal origin of adult diseases, suggesting that some adult diseases might be determined before birth by altered fetal development. Maternal diabetes subjects the fetus to an adverse environment that has been demonstrated to result in metabolic, cardiovascular and renal impairment in the offspring. The growing amount of obesity in young females in developed and some developing countries should contribute to increasing the incidence of diabetes among pregnant women. In this review, we discuss how renal and extrarenal mechanisms participate in the genesis of hypertension induced by a diabetic status during fetal development.
id ABDC-1_fc59ba88281fe8ac4d65e13997ddf250
oai_identifier_str oai:scielo:S0100-879X2011000900011
network_acronym_str ABDC-1
network_name_str Brazilian Journal of Medical and Biological Research
repository_id_str
spelling Prenatally programmed hypertension: role of maternal diabetesHypertensionMaternal diabetesRenal impairmentSodium retentionEpidemiological and experimental studies have led to the hypothesis of the fetal origin of adult diseases, suggesting that some adult diseases might be determined before birth by altered fetal development. Maternal diabetes subjects the fetus to an adverse environment that has been demonstrated to result in metabolic, cardiovascular and renal impairment in the offspring. The growing amount of obesity in young females in developed and some developing countries should contribute to increasing the incidence of diabetes among pregnant women. In this review, we discuss how renal and extrarenal mechanisms participate in the genesis of hypertension induced by a diabetic status during fetal development.Associação Brasileira de Divulgação Científica2011-09-01info:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersiontext/htmlhttp://old.scielo.br/scielo.php?script=sci_arttext&pid=S0100-879X2011000900011Brazilian Journal of Medical and Biological Research v.44 n.9 2011reponame:Brazilian Journal of Medical and Biological Researchinstname:Associação Brasileira de Divulgação Científica (ABDC)instacron:ABDC10.1590/S0100-879X2011007500109info:eu-repo/semantics/openAccessGomes,G.N.Gil,F.Z.eng2011-09-30T00:00:00Zoai:scielo:S0100-879X2011000900011Revistahttps://www.bjournal.org/https://old.scielo.br/oai/scielo-oai.phpbjournal@terra.com.br||bjournal@terra.com.br1414-431X0100-879Xopendoar:2011-09-30T00:00Brazilian Journal of Medical and Biological Research - Associação Brasileira de Divulgação Científica (ABDC)false
dc.title.none.fl_str_mv Prenatally programmed hypertension: role of maternal diabetes
title Prenatally programmed hypertension: role of maternal diabetes
spellingShingle Prenatally programmed hypertension: role of maternal diabetes
Gomes,G.N.
Hypertension
Maternal diabetes
Renal impairment
Sodium retention
title_short Prenatally programmed hypertension: role of maternal diabetes
title_full Prenatally programmed hypertension: role of maternal diabetes
title_fullStr Prenatally programmed hypertension: role of maternal diabetes
title_full_unstemmed Prenatally programmed hypertension: role of maternal diabetes
title_sort Prenatally programmed hypertension: role of maternal diabetes
author Gomes,G.N.
author_facet Gomes,G.N.
Gil,F.Z.
author_role author
author2 Gil,F.Z.
author2_role author
dc.contributor.author.fl_str_mv Gomes,G.N.
Gil,F.Z.
dc.subject.por.fl_str_mv Hypertension
Maternal diabetes
Renal impairment
Sodium retention
topic Hypertension
Maternal diabetes
Renal impairment
Sodium retention
description Epidemiological and experimental studies have led to the hypothesis of the fetal origin of adult diseases, suggesting that some adult diseases might be determined before birth by altered fetal development. Maternal diabetes subjects the fetus to an adverse environment that has been demonstrated to result in metabolic, cardiovascular and renal impairment in the offspring. The growing amount of obesity in young females in developed and some developing countries should contribute to increasing the incidence of diabetes among pregnant women. In this review, we discuss how renal and extrarenal mechanisms participate in the genesis of hypertension induced by a diabetic status during fetal development.
publishDate 2011
dc.date.none.fl_str_mv 2011-09-01
dc.type.driver.fl_str_mv info:eu-repo/semantics/article
dc.type.status.fl_str_mv info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.uri.fl_str_mv http://old.scielo.br/scielo.php?script=sci_arttext&pid=S0100-879X2011000900011
url http://old.scielo.br/scielo.php?script=sci_arttext&pid=S0100-879X2011000900011
dc.language.iso.fl_str_mv eng
language eng
dc.relation.none.fl_str_mv 10.1590/S0100-879X2011007500109
dc.rights.driver.fl_str_mv info:eu-repo/semantics/openAccess
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv text/html
dc.publisher.none.fl_str_mv Associação Brasileira de Divulgação Científica
publisher.none.fl_str_mv Associação Brasileira de Divulgação Científica
dc.source.none.fl_str_mv Brazilian Journal of Medical and Biological Research v.44 n.9 2011
reponame:Brazilian Journal of Medical and Biological Research
instname:Associação Brasileira de Divulgação Científica (ABDC)
instacron:ABDC
instname_str Associação Brasileira de Divulgação Científica (ABDC)
instacron_str ABDC
institution ABDC
reponame_str Brazilian Journal of Medical and Biological Research
collection Brazilian Journal of Medical and Biological Research
repository.name.fl_str_mv Brazilian Journal of Medical and Biological Research - Associação Brasileira de Divulgação Científica (ABDC)
repository.mail.fl_str_mv bjournal@terra.com.br||bjournal@terra.com.br
_version_ 1754302940286287872

Registros relacionados