Data on metabolic profile of insulin-degrading enzyme knockout mice

Detalhes bibliográficos
Autor(a) principal: Borges, D
Data de Publicação: 2019
Outros Autores: Meneses, M, Dias, T, Martins, F, Oliveira, PF, Alves, M, Macedo, M
Tipo de documento: Artigo
Idioma: eng
Título da fonte: Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos)
Texto Completo: https://hdl.handle.net/10216/139004
Resumo: Insulin-degrading enzyme (IDE) degrades and inactivates bioactive peptides such as insulin. As insulin is a master regulator of glucose homeostasis, lack of IDE is expected to have a profound impact on both insulin and glucose levels. This article shares data on glucose and insulin homeostasis of control, heterozygous and knockout mice for Ide after 18 weeks of a normal chow diet. This data article is related to a research article entitled “Knockout of insulin-degrading enzyme leads to mice testicular morphological changes and impaired sperm quality” (Meneses et al., 2019).
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spelling Data on metabolic profile of insulin-degrading enzyme knockout miceInsulin-degrading enzyme (IDE) degrades and inactivates bioactive peptides such as insulin. As insulin is a master regulator of glucose homeostasis, lack of IDE is expected to have a profound impact on both insulin and glucose levels. This article shares data on glucose and insulin homeostasis of control, heterozygous and knockout mice for Ide after 18 weeks of a normal chow diet. This data article is related to a research article entitled “Knockout of insulin-degrading enzyme leads to mice testicular morphological changes and impaired sperm quality” (Meneses et al., 2019).Elsevier20192019-01-01T00:00:00Zinfo:eu-repo/semantics/publishedVersioninfo:eu-repo/semantics/articleapplication/pdfhttps://hdl.handle.net/10216/139004eng2352-340910.1016/j.dib.2019.104023Borges, DMeneses, MDias, TMartins, FOliveira, PFAlves, MMacedo, Minfo:eu-repo/semantics/openAccessreponame:Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos)instname:Agência para a Sociedade do Conhecimento (UMIC) - FCT - Sociedade da Informaçãoinstacron:RCAAP2023-11-29T15:39:31Zoai:repositorio-aberto.up.pt:10216/139004Portal AgregadorONGhttps://www.rcaap.pt/oai/openaireopendoar:71602024-03-20T00:28:58.139574Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos) - Agência para a Sociedade do Conhecimento (UMIC) - FCT - Sociedade da Informaçãofalse
dc.title.none.fl_str_mv Data on metabolic profile of insulin-degrading enzyme knockout mice
title Data on metabolic profile of insulin-degrading enzyme knockout mice
spellingShingle Data on metabolic profile of insulin-degrading enzyme knockout mice
Borges, D
title_short Data on metabolic profile of insulin-degrading enzyme knockout mice
title_full Data on metabolic profile of insulin-degrading enzyme knockout mice
title_fullStr Data on metabolic profile of insulin-degrading enzyme knockout mice
title_full_unstemmed Data on metabolic profile of insulin-degrading enzyme knockout mice
title_sort Data on metabolic profile of insulin-degrading enzyme knockout mice
author Borges, D
author_facet Borges, D
Meneses, M
Dias, T
Martins, F
Oliveira, PF
Alves, M
Macedo, M
author_role author
author2 Meneses, M
Dias, T
Martins, F
Oliveira, PF
Alves, M
Macedo, M
author2_role author
author
author
author
author
author
dc.contributor.author.fl_str_mv Borges, D
Meneses, M
Dias, T
Martins, F
Oliveira, PF
Alves, M
Macedo, M
description Insulin-degrading enzyme (IDE) degrades and inactivates bioactive peptides such as insulin. As insulin is a master regulator of glucose homeostasis, lack of IDE is expected to have a profound impact on both insulin and glucose levels. This article shares data on glucose and insulin homeostasis of control, heterozygous and knockout mice for Ide after 18 weeks of a normal chow diet. This data article is related to a research article entitled “Knockout of insulin-degrading enzyme leads to mice testicular morphological changes and impaired sperm quality” (Meneses et al., 2019).
publishDate 2019
dc.date.none.fl_str_mv 2019
2019-01-01T00:00:00Z
dc.type.status.fl_str_mv info:eu-repo/semantics/publishedVersion
dc.type.driver.fl_str_mv info:eu-repo/semantics/article
format article
status_str publishedVersion
dc.identifier.uri.fl_str_mv https://hdl.handle.net/10216/139004
url https://hdl.handle.net/10216/139004
dc.language.iso.fl_str_mv eng
language eng
dc.relation.none.fl_str_mv 2352-3409
10.1016/j.dib.2019.104023
dc.rights.driver.fl_str_mv info:eu-repo/semantics/openAccess
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
dc.publisher.none.fl_str_mv Elsevier
publisher.none.fl_str_mv Elsevier
dc.source.none.fl_str_mv reponame:Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos)
instname:Agência para a Sociedade do Conhecimento (UMIC) - FCT - Sociedade da Informação
instacron:RCAAP
instname_str Agência para a Sociedade do Conhecimento (UMIC) - FCT - Sociedade da Informação
instacron_str RCAAP
institution RCAAP
reponame_str Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos)
collection Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos)
repository.name.fl_str_mv Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos) - Agência para a Sociedade do Conhecimento (UMIC) - FCT - Sociedade da Informação
repository.mail.fl_str_mv
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